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A patient with pancreatic hypoplasia was found to have an annular cardiac septal defect (ASD) with left-to-right shunting. The defects of the annulus and ventricular septum were associated with ductal-dependent hepatopulmonary vascular malformations that were similar in their surgical approach. This patient is the first with this syndrome to be followed-up long-term. The results of the persistent ductus arteriosus (PDA) ligation, anastomosis of the commissural vessel to the pulmonary artery and coarctationplasty were good. Six years after surgery, the patient shows no signs of recurrence of the ductal-dependent malformation.Gymnastics at the 1968 Summer Olympics – Men’s rings

The men’s rings competition at the 1968 Summer Olympics in Mexico City, Mexico, was held at the OlĂ­mpic Sports Center (Centro de los Deportes) on October 15.

Competition format

The gymnastics format continued to use the aggregation format. Each country entered a team of six gymnasts for competition. All entrants performed two routines: one on the apparatus of their choice and one on each of the remaining three apparatus. Scores from the four apparatus routines were added together with the scores from the individual all-around exercise to give a team total. No separate scores were recorded for qualification.

For the all-around qualification, all teams were entered with four gymnasts who performed only a floor exercise. The top six scores on that exercise were carried over to the all-around finals.

For each event, the four highest scoring gymnasts without regard to self-starts carried over their final result into the next round. Only two gymnasts per team were allowed to self-start. For the men’s rings final, the scores of the top six teams were summed and the lowest total was dropped. The third- and fourth-placed teams competed in the third place and fifth place finals for

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The A/J strain of mice (the Jackson laboratory strain), in contrast to the FVB/N strain (the NCTC strain) used for most other studies of mammary carcinogenesis, is highly sensitive to 2-amino-3-methylimidazo[4,5-f]quinoline (IQ), an environmental hepatocarcinogen. A/J mice undergo growth of premalignant, IQ-induced mammary tumors and malignant, IQ-induced tumors within 4 months after initiation with a single oral dose of 1 ppm IQ, whereas FVB/N mice fail to develop tumors in this time and concentration range. We compared the IQ-induced histopathology of IQ-induced mammary tumors from A/J and FVB/N mice. The A/J tumors showed histological evidence of differentiation, fewer abnormal mitotic figures, and less nuclear atypia than those from FVB/N mice. Also, the A/J tumors had a greater proportion of cells in the G(0)/G(1) phase of the cell cycle than those from FVB/N mice. We examined a panel of 33 “mammary-specific” genes to determine whether their transcriptional levels in mammary tumors from A/J mice compared with those from FVB/N mice could account for their respective differences in development. A/J mice had “higher levels” of 25 genes (listed in the right column of Table 1), including cyclin-D1, cyclin-E1, and cyclin-G2. We


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