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Macrophages are highly specialized cells in the innate immune system that are the first cells arriving at sites of infection and injury. They are considered to be heterogeneous, possessing variable states of activation, including classical pro-inflammatory macrophages (M1) and classical anti-inflammatory macrophages (M2). However, it is increasingly clear that this classification is not entirely accurate and that the most appropriate descriptor is the context-dependent and dynamic macrophage. In this review, we explore the extensive heterogeneity observed in macrophage subsets and propose that these represent different parts of a single spectrum, where state-dependent plasticity underlies the functional transitions between macrophages in health and disease.Status epilepticus and acute inflammatory demyelination.
The association between acute inflammatory demyelinating disorders and status epilepticus is well established. Status epilepticus is a common precipitating factor for the development of acute inflammatory demyelinating disorders, and patients with acute inflammatory demyelinating disorders are more likely to develop epilepsy than the general population. However, there are few data evaluating outcomes in these patients. Here we review the clinical features of status epilepticus in patients with acute inflammatory demyelinating disorders and describe the temporal profile of immune cell markers in patients with this association. Knowledge of the temporal profile of immune cell markers in this condition will assist in the diagnosis of patients with status epilepticus and further provide insights into their pathophysiology. It is hoped that this will aid to define the optimal therapeutic strategy for these patients in the future.Hepatic iron reduction in erythropoiesis-stimulating agents with rapidly and slowly produced iron chelators: experiments in rats and clinical studies in patients.
The present study aims to investigate the influence of the rate of iron removal on the clinical usefulness of erythropoiesis-stimulating agents. Fe2+-chelation by benzohydroxamate (BH2) and deferiprone (DFP) was compared. Fe2+ was induced by iron-loading of anemic rats. Chelation was expressed as the time course of Fe2+ plasma reduction. Iron-loading reduced BH2 iron removal in a linear and DFP in a stepwise fashion. Expressed as the percentage of Fe2+ reduction BH2 chelates
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